Zhang, X., P. Shan, S. Qureshi, R. Homer, R. Medzhitov, P. W. Noble, and P. J. Lee. 2005. Cutting edge: TLR4 deficiency confers susceptibility to lethal oxidant lung injury. J. Immunol. 175: 4834–4838 .

In Materials and Methods, in the first sentence under the heading Intranasal administration of recombinant adenovirus-containing HO-1 cDNA, the source for adenoviral HO-1 cDNA, the source for adenoviral HO-1 cDNA was incorrectly attributed. The source is stated in the corrected sentence below.

Mice were anesthetized with methoxyflurane, and then 5 × 108 PFU of adenoviral HO-1 (Ad-HO-1) (a gift from K. Kolls, University of Pittsburgh Medical Center, Pittsburgh, PA, and J. Alam, Alton Ochsner Medical Foundation, New Orleans, LA) (29) or adenoviral β-galactosidase (Ad-LacZ) (BD Biosciences) were adminstered intranasally to each mouse in a volume of 50 μl as described previously (12).

The authors also wish to add the reference shown below.

29. Otterbein, L. E., J. K. Kolls, L. L. Mantell, J. L. Cook, J. Alam, and A. M. K. Choi. 1999. Exogenous administration of heme oxygenase-1 by gene transfer provides protection against hyperoxia-induced lung injury. J. Clin. Invest. 103: 1047–1054.