Roan, F., T. A. Stoklasek, E. Whalen, J. A. Molitor, J. A. Bluestone, J. H. Buckner, and S. F. Ziegler. 2016. CD4+ group 1 innate lymphoid cells (ILC) form a functionally distinct ILC subset that is increased in systemic sclerosis. J. Immunol. 196: 2051–2062.
The graphs from Fig. 3B were inadvertently reversed in the published article. The upper graph corresponds to CD4− ILC1, while the lower graph corresponds to CD4+ ILC1. The corrected Fig. 3 is shown below. The figure legend is correct as published and is shown below for reference.
CD4+ and CD4− ILC1 cytokine production. Fresh PBMC were prepared from peripheral blood, lineage depleted using a MACS column, stained for ILC surface markers, and flow sorted for CD4+ and CD4− ILC1 subsets (n = 3) (A) or CXCR3+ and CXCR3− populations within the CD4+ and CD4− ILC1 subsets (n = 3) (B). The sorted populations were stimulated with PMA/ionomycin/brefeldin A for 6 h and then examined for intracellular cytokine expression by flow cytometry.
CD4+ and CD4− ILC1 cytokine production. Fresh PBMC were prepared from peripheral blood, lineage depleted using a MACS column, stained for ILC surface markers, and flow sorted for CD4+ and CD4− ILC1 subsets (n = 3) (A) or CXCR3+ and CXCR3− populations within the CD4+ and CD4− ILC1 subsets (n = 3) (B). The sorted populations were stimulated with PMA/ionomycin/brefeldin A for 6 h and then examined for intracellular cytokine expression by flow cytometry.
Additionally, the affiliation for the fourth author, Jerry A. Molitor, should be listed as follows: Division of Rheumatic and Autoimmune Diseases, Department of Medicine, University of Minnesota, Minneapolis, MN 55455.
