Singh, S. P., O. Huck, N. G. Abraham, and S. Amar. 2018. Kavain reduces Porphyromonas gingivalis–induced adipocyte inflammation: role of PGC-1α signaling. J. Immunol. 201: 1491–1499.
Due to an error in figure preparation, the wrong image was included for Fig. 6H. The corrected version of Fig. 6 is shown below. The figure legend was correct as published and is shown below for reference.
Effect of Kavain treatment on WT and PGC-1α–deficient 3T3-L1–derived adipocytes with and without exposure of P. gingivalis. Adiponectin is a protein produced by adipocytes and acts as a hormone. It has an important role in modulating glucose and lipid metabolism in insulin-sensitive tissues. Immunofluorescence was performed to detect the presence of adiponectin in (A) WT, (B) WT plus Kavain, (C) WT plus P. gingivalis, (D) WT plus P. gingivalis plus Kavain, (E) PGC-1α KD, (F) PGC-1α KD plus Kavain, (G) PGC-1α KD plus P. gingivalis, and (H) PGC-1α KD plus P. gingivalis plus Kavain. Original magnification ×100.
Effect of Kavain treatment on WT and PGC-1α–deficient 3T3-L1–derived adipocytes with and without exposure of P. gingivalis. Adiponectin is a protein produced by adipocytes and acts as a hormone. It has an important role in modulating glucose and lipid metabolism in insulin-sensitive tissues. Immunofluorescence was performed to detect the presence of adiponectin in (A) WT, (B) WT plus Kavain, (C) WT plus P. gingivalis, (D) WT plus P. gingivalis plus Kavain, (E) PGC-1α KD, (F) PGC-1α KD plus Kavain, (G) PGC-1α KD plus P. gingivalis, and (H) PGC-1α KD plus P. gingivalis plus Kavain. Original magnification ×100.
