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In (Fig. 3B in the original article, the panel for HDM-exposed Adamts12−/− mice was mistakenly duplicated from the WT panel. The correct KO image (from contemporaneous data) is shown in the corrected version of Fig. 3 below. The authors apologize for this error.
The figure legend for Fig. 3A and 3B has also been corrected to identify the arrow. The corrected figure legend is shown below.
Histological analysis of lung tissue after exposure. (A and B) Lung sections stained with H&E. Original magnification ×200. No inflammation was detected in peribronchial tissue of sham mice. After allergen exposure, WT mice and Adamts12−/− mice displayed a bronchial inflammation (arrow). Each image is representative of at least 10 animals. (C) Scoring of peribronchial inflammation after H&E staining in OVA-exposed inflammation model. **p < 0.01, ***p < 0.001, ANOVA test (n = 5–8/group). Similar results were obtained in two independent experiments. (D) Scoring of peribronchial inflammation after H&E staining in HDM-exposed inflammation model. *p < 0.05, ANOVA test (n = 5–8/group). Similar results were obtained in two independent experiments. (E) Eosinophil infiltration evidenced by Congo red staining. Original magnification ×400. Each image is representative of at least 10 animals. (F) Quantitative analysis of eosinophils in bronchial walls in OVA-exposed inflammation model. Six bronchi per mouse were quantified and results were expressed as number of eosinophils per millimeter of epithelial basement membrane. *p < 0.05, ***p < 0.001, ANOVA test (n = 5–8/group). Similar results were obtained in two independent experiments. (G) Quantitative analysis of eosinophils in bronchial walls in HDM-exposed inflammation model. Six bronchi per mouse were quantified and results were expressed as number of eosinophils per millimeter of epithelial basement membrane. *p < 0.05, ***p < 0.001, ANOVA test (n = 5–8/group). Similar results were obtained in two independent experiments.
Histological analysis of lung tissue after exposure. (A and B) Lung sections stained with H&E. Original magnification ×200. No inflammation was detected in peribronchial tissue of sham mice. After allergen exposure, WT mice and Adamts12−/− mice displayed a bronchial inflammation (arrow). Each image is representative of at least 10 animals. (C) Scoring of peribronchial inflammation after H&E staining in OVA-exposed inflammation model. **p < 0.01, ***p < 0.001, ANOVA test (n = 5–8/group). Similar results were obtained in two independent experiments. (D) Scoring of peribronchial inflammation after H&E staining in HDM-exposed inflammation model. *p < 0.05, ANOVA test (n = 5–8/group). Similar results were obtained in two independent experiments. (E) Eosinophil infiltration evidenced by Congo red staining. Original magnification ×400. Each image is representative of at least 10 animals. (F) Quantitative analysis of eosinophils in bronchial walls in OVA-exposed inflammation model. Six bronchi per mouse were quantified and results were expressed as number of eosinophils per millimeter of epithelial basement membrane. *p < 0.05, ***p < 0.001, ANOVA test (n = 5–8/group). Similar results were obtained in two independent experiments. (G) Quantitative analysis of eosinophils in bronchial walls in HDM-exposed inflammation model. Six bronchi per mouse were quantified and results were expressed as number of eosinophils per millimeter of epithelial basement membrane. *p < 0.05, ***p < 0.001, ANOVA test (n = 5–8/group). Similar results were obtained in two independent experiments.
