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Cloning and analysis of the promoter region of CCR5, a coreceptor for HIV-1 entry.

H Moriuchi, M Moriuchi, A S Fauci

J Immunol (1997) 159 (11): 5441–5449.

DOI: https://doi.org/10.4049/jimmunol.159.11.5441

Published: 01 December 1997

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Cloning and analysis of the promoter region of CXCR4, a coreceptor for HIV-1 entry.

M Moriuchi, H Moriuchi, W Turner, A S Fauci

J Immunol (1997) 159 (9): 4322–4329.

DOI: https://doi.org/10.4049/jimmunol.159.9.4322

Published: 01 November 1997

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Nuclear factor-kappa B potently up-regulates the promoter activity of RANTES, a chemokine that blocks HIV infection.

H Moriuchi, M Moriuchi, A S Fauci

J Immunol (1997) 158 (7): 3483–3491.

DOI: https://doi.org/10.4049/jimmunol.158.7.3483

Published: 01 April 1997

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Effect of Mycobacterium tuberculosis on HIV replication. Role of immune activation.

D Goletti, D Weissman, R W Jackson, N M Graham, D Vlahov, R S Klein, S S Munsiff, L Ortona, R Cauda, A S Fauci

J Immunol (1996) 157 (3): 1271–1278.

DOI: https://doi.org/10.4049/jimmunol.157.3.1271

Published: 01 August 1996

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Identification of multiple and distinct CD8+ T cell suppressor activities: dichotomy between infected and uninfected individuals, evolution with progression of disease, and sensitivity to gamma irradiation.

T D Barker, D Weissman, J A Daucher, K M Roche, A S Fauci

J Immunol (1996) 156 (11): 4476–4483.

DOI: https://doi.org/10.4049/jimmunol.156.11.4476

Published: 01 June 1996

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Modulation of endogenous IL-1 beta and IL-1 receptor antagonist results in opposing effects on HIV expression in chronically infected monocytic cells.

D Goletti, A L Kinter, E C Hardy, G Poli, A S Fauci

J Immunol (1996) 156 (9): 3501–3508.

DOI: https://doi.org/10.4049/jimmunol.156.9.3501

Published: 01 May 1996

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Both a precursor and a mature population of dendritic cells can bind HIV. However, only the mature population that expresses CD80 can pass infection to unstimulated CD4+ T cells.

D Weissman, Y Li, J M Orenstein, A S Fauci

J Immunol (1995) 155 (8): 4111–4117.

DOI: https://doi.org/10.4049/jimmunol.155.8.4111

Published: 15 October 1995

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Analysis of apoptosis in lymph nodes of HIV-infected persons. Intensity of apoptosis correlates with the general state of activation of the lymphoid tissue and not with stage of disease or viral burden.

C A Muro-Cacho, G Pantaleo, A S Fauci

J Immunol (1995) 154 (10): 5555–5566.

DOI: https://doi.org/10.4049/jimmunol.154.10.5555

Published: 15 May 1995

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HIV replication in IL-2-stimulated peripheral blood mononuclear cells is driven in an autocrine/paracrine manner by endogenous cytokines.

A L Kinter, G Poli, L Fox, E Hardy, A S Fauci

J Immunol (1995) 154 (5): 2448–2459.

DOI: https://doi.org/10.4049/jimmunol.154.5.2448

Published: 01 March 1995

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CD34+ bone marrow cells are infected with HIV in a subset of seropositive individuals.

S K Stanley, S W Kessler, J S Justement, S M Schnittman, J J Greenhouse, C C Brown, L Musongela, K Musey, B Kapita, A S Fauci

J Immunol (1992) 149 (2): 689–697.

DOI: https://doi.org/10.4049/jimmunol.149.2.689

Published: 15 July 1992

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Recombinant gp120 specifically enhances tumor necrosis factor-alpha production and Ig secretion in B lymphocytes from HIV-infected individuals but not from seronegative donors.

P Rieckmann, G Poli, C H Fox, J H Kehrl, A S Fauci

J Immunol (1991) 147 (9): 2922–2927.

DOI: https://doi.org/10.4049/jimmunol.147.9.2922

Published: 01 November 1991

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Thymic microenvironment induces HIV expression. Physiologic secretion of IL-6 by thymic epithelial cells up-regulates virus expression in chronically infected cells.

S M Schnittman, K H Singer, J J Greenhouse, S K Stanley, L P Whichard, P T Le, B F Haynes, A S Fauci

J Immunol (1991) 147 (8): 2553–2558.

DOI: https://doi.org/10.4049/jimmunol.147.8.2553

Published: 15 October 1991

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Anti-CD2 receptor antibodies activate the HIV long terminal repeat in T lymphocytes.

P Bressler, G Pantaleo, A Demaria, A S Fauci

J Immunol (1991) 147 (7): 2290–2294.

DOI: https://doi.org/10.4049/jimmunol.147.7.2290

Published: 01 October 1991

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Transforming growth factor-beta suppresses human B lymphocyte Ig production by inhibiting synthesis and the switch from the membrane form to the secreted form of Ig mRNA.

J H Kehrl, C Thevenin, P Rieckmann, A S Fauci

J Immunol (1991) 146 (11): 4016–4023.

DOI: https://doi.org/10.4049/jimmunol.146.11.4016

Published: 01 June 1991

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IL-6 and tumor necrosis factor-alpha. Autocrine and paracrine cytokines involved in B cell function.

P Rieckmann, F D'Alessandro, R P Nordan, A S Fauci, J H Kehrl

J Immunol (1991) 146 (10): 3462–3468.

DOI: https://doi.org/10.4049/jimmunol.146.10.3462

Published: 15 May 1991

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Complement subcomponent C1q stimulates Ig production by human B lymphocytes.

K R Young, Jr, J L Ambrus, Jr, A Malbran, A S Fauci, A J Tenner

J Immunol (1991) 146 (10): 3356–3364.

DOI: https://doi.org/10.4049/jimmunol.146.10.3356

Published: 15 May 1991

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Infection of CD8+ T lymphocytes with HIV. Requirement for interaction with infected CD4+ cells and induction of infectious virus from chronically infected CD8+ cells.

A De Maria, G Pantaleo, S M Schnittman, J J Greenhouse, M Baseler, J M Orenstein, A S Fauci

J Immunol (1991) 146 (7): 2220–2226.

DOI: https://doi.org/10.4049/jimmunol.146.7.2220

Published: 01 April 1991

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Functional studies examining the subpopulation of human B lymphocytes responding to high molecular weight B cell growth factor.

J L Ambrus, Jr, L Chesky, D Stephany, P McFarland, H Mostowski, A S Fauci

J Immunol (1990) 145 (12): 3949–3955.

DOI: https://doi.org/10.4049/jimmunol.145.12.3949

Published: 15 December 1990

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Heat shock induction of HIV production from chronically infected promonocytic and T cell lines.

S K Stanley, P B Bressler, G Poli, A S Fauci

J Immunol (1990) 145 (4): 1120–1126.

DOI: https://doi.org/10.4049/jimmunol.145.4.1120

Published: 15 August 1990

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In vivo T lymphocyte origin of macrophage-tropic strains of HIV. Role of monocytes during in vitro isolation and in vivo infection.

F E Massari, G Poli, S M Schnittman, M C Psallidopoulos, V Davey, A S Fauci

J Immunol (1990) 144 (12): 4628–4632.

DOI: https://doi.org/10.4049/jimmunol.144.12.4628

Published: 15 June 1990

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NARROW

Cloning and analysis of the promoter region of CCR5, a coreceptor for HIV-1 entry.

Cloning and analysis of the promoter region of CXCR4, a coreceptor for HIV-1 entry.

Nuclear factor-kappa B potently up-regulates the promoter activity of RANTES, a chemokine that blocks HIV infection.

Effect of Mycobacterium tuberculosis on HIV replication. Role of immune activation.

Identification of multiple and distinct CD8+ T cell suppressor activities: dichotomy between infected and uninfected individuals, evolution with progression of disease, and sensitivity to gamma irradiation.

Modulation of endogenous IL-1 beta and IL-1 receptor antagonist results in opposing effects on HIV expression in chronically infected monocytic cells.

Both a precursor and a mature population of dendritic cells can bind HIV. However, only the mature population that expresses CD80 can pass infection to unstimulated CD4+ T cells.

Analysis of apoptosis in lymph nodes of HIV-infected persons. Intensity of apoptosis correlates with the general state of activation of the lymphoid tissue and not with stage of disease or viral burden.

HIV replication in IL-2-stimulated peripheral blood mononuclear cells is driven in an autocrine/paracrine manner by endogenous cytokines.

CD34+ bone marrow cells are infected with HIV in a subset of seropositive individuals.

Recombinant gp120 specifically enhances tumor necrosis factor-alpha production and Ig secretion in B lymphocytes from HIV-infected individuals but not from seronegative donors.

Thymic microenvironment induces HIV expression. Physiologic secretion of IL-6 by thymic epithelial cells up-regulates virus expression in chronically infected cells.

Anti-CD2 receptor antibodies activate the HIV long terminal repeat in T lymphocytes.

Transforming growth factor-beta suppresses human B lymphocyte Ig production by inhibiting synthesis and the switch from the membrane form to the secreted form of Ig mRNA.

IL-6 and tumor necrosis factor-alpha. Autocrine and paracrine cytokines involved in B cell function.

Complement subcomponent C1q stimulates Ig production by human B lymphocytes.

Infection of CD8+ T lymphocytes with HIV. Requirement for interaction with infected CD4+ cells and induction of infectious virus from chronically infected CD8+ cells.

Functional studies examining the subpopulation of human B lymphocytes responding to high molecular weight B cell growth factor.

Heat shock induction of HIV production from chronically infected promonocytic and T cell lines.

In vivo T lymphocyte origin of macrophage-tropic strains of HIV. Role of monocytes during in vitro isolation and in vivo infection.

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