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Amy G. Hise
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Journal Articles

Neutrophil Caspase-11 Is Required for Cleavage of Caspase-1 and Secretion of IL-1β in Aspergillus fumigatus Infection

Yan Sun, Serena Abbondante, Mausita Karmakar, Steven de Jesus Carrion, Chengye Che, Amy G. Hise, Eric Pearlman
Journal: The Journal of Immunology
J Immunol (2018) 201 (9): 2767–2775.
https://doi.org/10.4049/jimmunol.1701195
Published: 01 November 2018
View article titled, Neutrophil Caspase-11 Is Required for Cleavage of Caspase-1 and Secretion of IL-1β in <em>Aspergillus fumigatus</em> Infection
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Includes: Supplementary data
Journal Articles

Neutrophil IL-1β Processing Induced by Pneumolysin Is Mediated by the NLRP3/ASC Inflammasome and Caspase-1 Activation and Is Dependent on K+ Efflux

Mausita Karmakar, Michael Katsnelson, Hesham A. Malak, Neil G. Greene, Scott J. Howell, Amy G. Hise, Andrew Camilli, Aras Kadioglu, George R. Dubyak, Eric Pearlman
Journal: The Journal of Immunology
J Immunol (2015) 194 (4): 1763–1775.
https://doi.org/10.4049/jimmunol.1401624
Published: 15 February 2015
View article titled, Neutrophil IL-1β Processing Induced by Pneumolysin Is Mediated by the NLRP3/ASC Inflammasome and Caspase-1 Activation and Is Dependent on K<sup>+</sup> Efflux
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Includes: Multimedia, Supplementary data
Journal Articles

β-Defensin 1 Plays a Role in Acute Mucosal Defense against Candida albicans

Jeffrey Tomalka, Elaheh Azodi, Hema P. Narra, Krupen Patel, Samantha O’Neill, Cisley Cardwell, Brian A. Hall, James M. Wilson, Amy G. Hise
Journal: The Journal of Immunology
J Immunol (2015) 194 (4): 1788–1795.
https://doi.org/10.4049/jimmunol.1203239
Published: 15 February 2015
View article titled, β-Defensin 1 Plays a Role in Acute Mucosal Defense against <em>Candida albicans</em>
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Includes: Supplementary data
Journal Articles

Caspase-8 Modulates Dectin-1 and Complement Receptor 3–Driven IL-1β Production in Response to β-Glucans and the Fungal Pathogen, Candida albicans

Sandhya Ganesan, Vijay A. K. Rathinam, Lukas Bossaller, Kelly Army, William J. Kaiser, Edward S. Mocarski, Christopher P. Dillon, Douglas R. Green, Tanya N. Mayadas, Stuart M. Levitz, Amy G. Hise, Neal Silverman, Katherine A. Fitzgerald
Journal: The Journal of Immunology
J Immunol (2014) 193 (5): 2519–2530.
https://doi.org/10.4049/jimmunol.1400276
Published: 01 September 2014
View article titled, Caspase-8 Modulates Dectin-1 and Complement Receptor 3–Driven IL-1β Production in Response to β-Glucans and the Fungal Pathogen, <em>Candida albicans</em>
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Includes: Supplementary data
Journal Articles

Cutting Edge: IL-1β Processing during Pseudomonas aeruginosa Infection Is Mediated by Neutrophil Serine Proteases and Is Independent of NLRC4 and Caspase-1

Mausita Karmakar, Yan Sun, Amy G. Hise, Arne Rietsch, Eric Pearlman
Journal: The Journal of Immunology
J Immunol (2012) 189 (9): 4231–4235.
https://doi.org/10.4049/jimmunol.1201447
Published: 01 November 2012
View article titled, Cutting Edge: IL-1β Processing during <em>Pseudomonas aeruginosa</em> Infection Is Mediated by Neutrophil Serine Proteases and Is Independent of NLRC4 and Caspase-1
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Includes: Supplementary data
Journal Articles

Innate Immune Responses to Endosymbiotic Wolbachia Bacteria in Brugia malayi and Onchocerca volvulus Are Dependent on TLR2, TLR6, MyD88, and Mal, but Not TLR4, TRIF, or TRAM

Amy G. Hise, Katrin Daehnel, Illona Gillette-Ferguson, Eun Cho, Helen F. McGarry, Mark J. Taylor, Douglas T. Golenbock, Katherine A. Fitzgerald, James W. Kazura, Eric Pearlman
Journal: The Journal of Immunology
J Immunol (2007) 178 (2): 1068–1076.
https://doi.org/10.4049/jimmunol.178.2.1068
Published: 15 January 2007
View article titled, Innate Immune Responses to Endosymbiotic <em>Wolbachia</em> Bacteria in <em>Brugia malayi</em> and <em>Onchocerca volvulus</em> Are Dependent on TLR2, TLR6, MyD88, and Mal, but Not TLR4, TRIF, or TRAM
Open the PDF for Innate Immune Responses to Endosymbiotic <em>Wolbachia</em> Bacteria in <em>Brugia malayi</em> and <em>Onchocerca volvulus</em> Are Dependent on TLR2, TLR6, MyD88, and Mal, but Not TLR4, TRIF, or TRAM in another window
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