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Arne Yndestad
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Journal Articles

Increased Complement Factor B and Bb Levels Are Associated with Mortality in Patients with Severe Aortic Stenosis

Negar Shahini, Thor Ueland, Andreas Auensen, Annika E. Michelsen, Judith K. Ludviksen, Amjad I. Hussain, Kjell I. Pettersen, Svend Aakhus, Torvald Espeland, Ida G. Lunde, Michael Kirschfink, Per H. Nilsson, Tom Eirik Mollnes, Lars Gullestad, Pål Aukrust, Arne Yndestad, Mieke C. Louwe
Journal: The Journal of Immunology
J Immunol (2019) 203 (7): 1973–1980.
https://doi.org/10.4049/jimmunol.1801244
Published: 01 October 2019
View article titled, Increased Complement Factor B and Bb Levels Are Associated with Mortality in Patients with Severe Aortic Stenosis
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Includes: Supplementary data
Journal Articles

Low Cellular NAD+ Compromises Lipopolysaccharide-Induced Inflammatory Responses via Inhibiting TLR4 Signal Transduction in Human Monocytes

Kuan Yang, Knut Husø Lauritzen, Maria Belland Olsen, Tuva Børresdatter Dahl, Trine Ranheim, Mohammed Shakil Ahmed, Håvard Attramadal, Pål Aukrust, Bente Halvorsen, Tuula Anneli Nyman, Øystein Sandanger, Arne Yndestad
Journal: The Journal of Immunology
J Immunol (2019) 203 (6): 1598–1608.
https://doi.org/10.4049/jimmunol.1801382
Published: 15 September 2019
View article titled, Low Cellular NAD<sup>+</sup> Compromises Lipopolysaccharide-Induced Inflammatory Responses via Inhibiting TLR4 Signal Transduction in Human Monocytes
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Includes: Supplementary data
Journal Articles

Increased Levels of Soluble CD40L in African Tick Bite Fever: Possible Involvement of TLRs in the Pathogenic Interaction between Rickettsia africae, Endothelial Cells, and Platelets

Jan K. Damås, Mogens Jensenius, Thor Ueland, Kari Otterdal, Arne Yndestad, Stig S. Frøland, Jean-Marc Rolain, Bjørn Myrvang, Didier Raoult, Pål Aukrust
Journal: The Journal of Immunology
J Immunol (2006) 177 (4): 2699–2706.
https://doi.org/10.4049/jimmunol.177.4.2699
Published: 15 August 2006
View article titled, Increased Levels of Soluble CD40L in African Tick Bite Fever: Possible Involvement of TLRs in the Pathogenic Interaction between <em>Rickettsia africae</em>, Endothelial Cells, and Platelets
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