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György Haskó
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Journal Articles

Ecto-5′-nucleotidase (CD73) decreases mortality and organ injury in sepsis (56.31)

Zoltán Németh, Balázs Csóka, Balázs Koscsó, Zoltán Spolarics, Rolando Rolandelli, György Haskó
Journal: The Journal of Immunology
J Immunol (2011) 186 (1_Supplement): 56.31.
https://doi.org/10.4049/jimmunol.186.Supp.56.31
Published: 01 April 2011
View articletitled, Ecto-5′-nucleotidase (CD73) decreases mortality and organ injury in sepsis (56.31)
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Journal Articles

Adenosine A2A Receptor Inactivation Increases Survival in Polymicrobial Sepsis

Zoltán H. Németh, Balázs Csóka, Jeanette Wilmanski, DaZhong Xu, Qi Lu, Catherine Ledent, Edwin A. Deitch, Pál Pacher, Zoltán Spolarics, György Haskó
Journal: The Journal of Immunology
J Immunol (2006) 176 (9): 5616–5626.
https://doi.org/10.4049/jimmunol.176.9.5616
Published: 01 May 2006
View articletitled, Adenosine A2A Receptor Inactivation Increases Survival in Polymicrobial Sepsis
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Includes: Supplementary data
Journal Articles

Adenosine Augments IL-10 Production by Macrophages through an A2B Receptor-Mediated Posttranscriptional Mechanism

Zoltán H. Németh, Carol S. Lutz, Balázs Csóka, Edwin A. Deitch, S. Joseph Leibovich, William C. Gause, Masahide Tone, Pál Pacher, E. Sylvester Vizi, György Haskó
Journal: The Journal of Immunology
J Immunol (2005) 175 (12): 8260–8270.
https://doi.org/10.4049/jimmunol.175.12.8260
Published: 15 December 2005
View articletitled, Adenosine Augments IL-10 Production by Macrophages through an A2B Receptor-Mediated Posttranscriptional Mechanism
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Journal Articles

Inosine Inhibits Inflammatory Cytokine Production by a Posttranscriptional Mechanism and Protects Against Endotoxin-Induced Shock

György Haskó, David G. Kuhel, Zoltán H. Németh, Jon G. Mabley, Robert F. Stachlewitz, László Virág, Zsolt Lohinai, Garry J. Southan, Andrew L. Salzman, Csaba Szabó
Journal: The Journal of Immunology
J Immunol (2000) 164 (2): 1013–1019.
https://doi.org/10.4049/jimmunol.164.2.1013
Published: 15 January 2000
View articletitled, Inosine Inhibits Inflammatory Cytokine Production by a Posttranscriptional Mechanism and Protects Against Endotoxin-Induced Shock
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