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Subunits of IgM Reconstitute Defective Contact Sensitivity in B-1 Cell-Deficient xid Mice: κ Light Chains Recruit T Cells Independent of Complement

Vipin Paliwal, Ryohei F. Tsuji, Marian Szczepanik, Ivana Kawikova, Regis A. Campos, Manfred Kneilling, Martin Röcken, Janine Schuurman, Frank A. Redegeld, Frans P. Nijkamp, Philip W. Askenase

J Immunol (2002) 169 (8): 4113–4123.

https://doi.org/10.4049/jimmunol.169.8.4113

Published: 15 October 2002

View article titled, Subunits of IgM Reconstitute Defective Contact Sensitivity in B-1 Cell-Deficient xid Mice: κ Light Chains Recruit T Cells Independent of Complement

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Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4⁺ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions

Pascal Launois, Alain Gumy, Hayo Himmelrich, Richard M. Locksley, Martin Röcken, Jacques A. Louis

J Immunol (2002) 168 (9): 4628–4635.

https://doi.org/10.4049/jimmunol.168.9.4628

Published: 01 May 2002

View article titled, Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4+ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions

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In BALB/c Mice, IL-4 Production During the Initial Phase of Infection with Leishmania major Is Necessary and Sufficient to Instruct Th2 Cell Development Resulting in Progressive Disease

Hayo Himmelrich, Pascal Launois, Ivan Maillard, Tilo Biedermann, Fabienne Tacchini-Cottier, Richard M. Locksley, Martin Röcken, Jacques A. Louis

J Immunol (2000) 164 (9): 4819–4825.

https://doi.org/10.4049/jimmunol.164.9.4819

Published: 01 May 2000

View article titled, In BALB/c Mice, IL-4 Production During the Initial Phase of Infection with Leishmania major Is Necessary and Sufficient to Instruct Th2 Cell Development Resulting in Progressive Disease

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Cutting Edge: CpG Oligodeoxynucleotides Trigger Protective and Curative Th1 Responses in Lethal Murine Leishmaniasis

Stefan Zimmermann, Oliver Egeter, Susanne Hausmann, Grayson B. Lipford, Martin Röcken, Hermann Wagner, Klaus Heeg

J Immunol (1998) 160 (8): 3627–3630.

https://doi.org/10.4049/jimmunol.160.8.3627

Published: 15 April 1998

View article titled, Cutting Edge: CpG Oligodeoxynucleotides Trigger Protective and Curative Th1 Responses in Lethal Murine Leishmaniasis

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Subunits of IgM Reconstitute Defective Contact Sensitivity in B-1 Cell-Deficient xid Mice: κ Light Chains Recruit T Cells Independent of Complement

Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4⁺ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions

In BALB/c Mice, IL-4 Production During the Initial Phase of Infection with Leishmania major Is Necessary and Sufficient to Instruct Th2 Cell Development Resulting in Progressive Disease

Cutting Edge: CpG Oligodeoxynucleotides Trigger Protective and Curative Th1 Responses in Lethal Murine Leishmaniasis

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Subunits of IgM Reconstitute Defective Contact Sensitivity in B-1 Cell-Deficient xid Mice: κ Light Chains Recruit T Cells Independent of Complement

Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4+ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions

In BALB/c Mice, IL-4 Production During the Initial Phase of Infection with Leishmania major Is Necessary and Sufficient to Instruct Th2 Cell Development Resulting in Progressive Disease

Cutting Edge: CpG Oligodeoxynucleotides Trigger Protective and Curative Th1 Responses in Lethal Murine Leishmaniasis

Email alerts

Publications

Submit a Manuscript

General Information

Journal Services

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Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4⁺ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions