Page 1 | Search Results | The Journal of Immunology | American Association of Immunologists

1-7 of 7

Sort by

Sort Order Select

Journal Articles

Regulatory B Cells Shape the Development of Th2 Immune Responses in BALB/c Mice Infected with Leishmania major Through IL-10 Production

Catherine Ronet, Yazmin Hauyon-La Torre, Mélanie Revaz-Breton, Béatris Mastelic, Fabienne Tacchini-Cottier, Jacques Louis, Pascal Launois

J Immunol (2010) 184 (2): 886–894.

https://doi.org/10.4049/jimmunol.0901114

Published: 27 December 2009

View article titled, Regulatory B Cells Shape the Development of Th2 Immune Responses in BALB/c Mice Infected with Leishmania major Through IL-10 Production

Add to Citation Manager

Journal Articles

Leishmania major-Specific B Cells Are Necessary for Th2 Cell Development and Susceptibility to L. major LV39 in BALB/c Mice

Catherine Ronet, Heike Voigt, Hayo Himmelrich, Marie-Agnès Doucey, Yazmin Hauyon-La Torre, Mélanie Revaz-Breton, Fabienne Tacchini-Cottier, Claude Bron, Jacques Louis, Pascal Launois

J Immunol (2008) 180 (7): 4825–4835.

https://doi.org/10.4049/jimmunol.180.7.4825

Published: 01 April 2008

View article titled, Leishmania major-Specific B Cells Are Necessary for Th2 Cell Development and Susceptibility to L. major LV39 in BALB/c Mice

Add to Citation Manager

Journal Articles

Macrophages Induce Neutrophil Apoptosis through Membrane TNF, a Process Amplified by Leishmania major

Cindy Allenbach, Christel Zufferey, Cynthia Perez, Pascal Launois, Christoph Mueller, Fabienne Tacchini-Cottier

J Immunol (2006) 176 (11): 6656–6664.

https://doi.org/10.4049/jimmunol.176.11.6656

Published: 01 June 2006

View article titled, Macrophages Induce Neutrophil Apoptosis through Membrane TNF, a Process Amplified by Leishmania major

Add to Citation Manager

Journal Articles

The Early IL-4 Response to Leishmania major and the Resulting Th2 Cell Maturation Steering Progressive Disease in BALB/c Mice Are Subject to the Control of Regulatory CD4⁺CD25⁺ T Cells

Abraham Aseffa, Alain Gumy, Pascal Launois, H. Robson MacDonald, Jacques A. Louis, Fabienne Tacchini-Cottier

J Immunol (2002) 169 (6): 3232–3241.

https://doi.org/10.4049/jimmunol.169.6.3232

Published: 15 September 2002

View article titled, The Early IL-4 Response to Leishmania major and the Resulting Th2 Cell Maturation Steering Progressive Disease in BALB/c Mice Are Subject to the Control of Regulatory CD4+CD25+ T Cells

Add to Citation Manager

Journal Articles

Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4⁺ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions

Pascal Launois, Alain Gumy, Hayo Himmelrich, Richard M. Locksley, Martin Röcken, Jacques A. Louis

J Immunol (2002) 168 (9): 4628–4635.

https://doi.org/10.4049/jimmunol.168.9.4628

Published: 01 May 2002

View article titled, Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4+ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions

Add to Citation Manager

Journal Articles

In BALB/c Mice, IL-4 Production During the Initial Phase of Infection with Leishmania major Is Necessary and Sufficient to Instruct Th2 Cell Development Resulting in Progressive Disease

Hayo Himmelrich, Pascal Launois, Ivan Maillard, Tilo Biedermann, Fabienne Tacchini-Cottier, Richard M. Locksley, Martin Röcken, Jacques A. Louis

J Immunol (2000) 164 (9): 4819–4825.

https://doi.org/10.4049/jimmunol.164.9.4819

Published: 01 May 2000

View article titled, In BALB/c Mice, IL-4 Production During the Initial Phase of Infection with Leishmania major Is Necessary and Sufficient to Instruct Th2 Cell Development Resulting in Progressive Disease

Add to Citation Manager

Journal Articles

The IL-4 Rapidly Produced in BALB/c Mice After Infection with Leishmania major Down-Regulates IL-12 Receptor β2-Chain Expression on CD4⁺ T Cells Resulting in a State of Unresponsiveness to IL-12

Hayo Himmelrich, Carlos Parra-Lopez, Fabienne Tacchini-Cottier, Jacques A. Louis, Pascal Launois

J Immunol (1998) 161 (11): 6156–6163.

https://doi.org/10.4049/jimmunol.161.11.6156

Published: 01 December 1998

View article titled, The IL-4 Rapidly Produced in BALB/c Mice After Infection with Leishmania major Down-Regulates IL-12 Receptor β2-Chain Expression on CD4+ T Cells Resulting in a State of Unresponsiveness to IL-12

Add to Citation Manager

NARROW

Regulatory B Cells Shape the Development of Th2 Immune Responses in BALB/c Mice Infected with Leishmania major Through IL-10 Production

Leishmania major-Specific B Cells Are Necessary for Th2 Cell Development and Susceptibility to L. major LV39 in BALB/c Mice

Macrophages Induce Neutrophil Apoptosis through Membrane TNF, a Process Amplified by Leishmania major

The Early IL-4 Response to Leishmania major and the Resulting Th2 Cell Maturation Steering Progressive Disease in BALB/c Mice Are Subject to the Control of Regulatory CD4⁺CD25⁺ T Cells

Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4⁺ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions

In BALB/c Mice, IL-4 Production During the Initial Phase of Infection with Leishmania major Is Necessary and Sufficient to Instruct Th2 Cell Development Resulting in Progressive Disease

The IL-4 Rapidly Produced in BALB/c Mice After Infection with Leishmania major Down-Regulates IL-12 Receptor β2-Chain Expression on CD4⁺ T Cells Resulting in a State of Unresponsiveness to IL-12

Email alerts

Publications

Submit a Manuscript

General Information

Journal Services

Regulatory B Cells Shape the Development of Th2 Immune Responses in BALB/c Mice Infected with Leishmania major Through IL-10 Production

Leishmania major-Specific B Cells Are Necessary for Th2 Cell Development and Susceptibility to L. major LV39 in BALB/c Mice

Macrophages Induce Neutrophil Apoptosis through Membrane TNF, a Process Amplified by Leishmania major

The Early IL-4 Response to Leishmania major and the Resulting Th2 Cell Maturation Steering Progressive Disease in BALB/c Mice Are Subject to the Control of Regulatory CD4+CD25+ T Cells

Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4+ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions

In BALB/c Mice, IL-4 Production During the Initial Phase of Infection with Leishmania major Is Necessary and Sufficient to Instruct Th2 Cell Development Resulting in Progressive Disease

The IL-4 Rapidly Produced in BALB/c Mice After Infection with Leishmania major Down-Regulates IL-12 Receptor β2-Chain Expression on CD4+ T Cells Resulting in a State of Unresponsiveness to IL-12

Email alerts

Publications

Submit a Manuscript

General Information

Journal Services

This Feature Is Available To Subscribers Only

The Early IL-4 Response to Leishmania major and the Resulting Th2 Cell Maturation Steering Progressive Disease in BALB/c Mice Are Subject to the Control of Regulatory CD4⁺CD25⁺ T Cells

Rapid IL-4 Production by Leishmania Homolog of Mammalian RACK1-Reactive CD4⁺ T Cells in Resistant Mice Treated Once with Anti-IL-12 or -IFN-γ Antibodies at the Onset of Infection with Leishmania major Instructs Th2 Cell Development, Resulting in Nonhealing Lesions

The IL-4 Rapidly Produced in BALB/c Mice After Infection with Leishmania major Down-Regulates IL-12 Receptor β2-Chain Expression on CD4⁺ T Cells Resulting in a State of Unresponsiveness to IL-12