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Commensal microbiota induces IL-17A-producing innate lymphoid cells that augment skin inflammation in cutaneous leishmaniasis

TEJ PRATAP SINGH, Augusto M Carvalho, Elizabeth A Grice, Phillip Scott

J Immunol (2021) 206 (1_Supplement): 99.07.

DOI: https://doi.org/10.4049/jimmunol.206.Supp.99.07

Published: 01 May 2021

View article titled, Commensal microbiota induces IL-17A-producing innate lymphoid cells that augment skin inflammation in cutaneous leishmaniasis

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A systemic inflammatory response primes blood monocytes in cutaneous leishmaniasis

Camila Farias Amorim, Brukawit A Goshu, Cláudia Lombana, Fernanda O Novais, Ba T. Nguyen, Lucas P. Carvalho, Edgar M. Carvalho, Daniel P. Beiting, Phillip Scott

J Immunol (2021) 206 (1_Supplement): 99.15.

DOI: https://doi.org/10.4049/jimmunol.206.Supp.99.15

Published: 01 May 2021

View article titled, A systemic inflammatory response primes blood monocytes in cutaneous leishmaniasis

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MALNUTRITION IMPACTS PROTECTIVE IMMUNE RESPONSES AND MUCOSAL IMMUNITY DURING VISCERAL LEISHMANIASIS

Laís Amorim Sacramento, Cláudia Lombana, Phillip Scott

J Immunol (2021) 206 (1_Supplement): 16.19.

DOI: https://doi.org/10.4049/jimmunol.206.Supp.16.19

Published: 01 May 2021

View article titled, MALNUTRITION IMPACTS PROTECTIVE IMMUNE RESPONSES AND MUCOSAL IMMUNITY DURING VISCERAL LEISHMANIASIS

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Journal Articles

CD8⁺ T Cells Lack Local Signals To Produce IFN-γ in the Skin during Leishmania Infection

Fernanda O. Novais, Andrea C. Wong, Daniel O. Villareal, Daniel P. Beiting, Phillip Scott

J Immunol (2018) 200 (5): 1737–1745.

DOI: https://doi.org/10.4049/jimmunol.1701597

Published: 01 March 2018

View article titled, CD8+ T Cells Lack Local Signals To Produce IFN-γ in the Skin during Leishmania Infection

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Includes: Supplementary data

Journal Articles

Leishmania major Infection–Induced VEGF-A/VEGFR-2 Signaling Promotes Lymphangiogenesis That Controls Disease

Tiffany Weinkopff, Christoph Konradt, David A. Christian, Dennis E. Discher, Christopher A. Hunter, Phillip Scott

J Immunol (2016) 197 (5): 1823–1831.

DOI: https://doi.org/10.4049/jimmunol.1600717

Published: 01 September 2016

View article titled, Leishmania major Infection–Induced VEGF-A/VEGFR-2 Signaling Promotes Lymphangiogenesis That Controls Disease

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Includes: Multimedia, Supplementary data

Journal Articles

Lymphocytic Choriomeningitis Virus Expands a Population of NKG2D⁺CD8⁺ T Cells That Exacerbates Disease in Mice Coinfected with Leishmania major

Erika J. Crosby, Megan Clark, Fernanda O. Novais, E. John Wherry, Phillip Scott

J Immunol (2015) 195 (7): 3301–3310.

DOI: https://doi.org/10.4049/jimmunol.1500855

Published: 01 October 2015

View article titled, Lymphocytic Choriomeningitis Virus Expands a Population of NKG2D+CD8+ T Cells That Exacerbates Disease in Mice Coinfected with Leishmania major

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Journal Articles

Bystander CD8 T cells produce granzyme B and promote exacerbated disease during Leishmania major infection (P3079)

Erika Crosby, E. Wherry, Phillip Scott

J Immunol (2013) 190 (1_Supplement): 187.13.

DOI: https://doi.org/10.4049/jimmunol.190.Supp.187.13

Published: 01 May 2013

View article titled, Bystander CD8 T cells produce granzyme B and promote exacerbated disease during Leishmania major infection (P3079)

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Journal Articles

Influence of viral specific memory cells on Leishmania major infection (164.12)

Erika Crosby, E. Wherry, Phillip Scott

J Immunol (2012) 188 (1_Supplement): 164.12.

DOI: https://doi.org/10.4049/jimmunol.188.Supp.164.12

Published: 01 May 2012

View article titled, Influence of viral specific memory cells on Leishmania major infection (164.12)

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A mixed Th1/Th2 response induces optimal resistance to Leishmania braziliensis (43.21)

Fernanda Novais, James Lee, Camila de Oliveira, Phillip Scott

J Immunol (2012) 188 (1_Supplement): 43.21.

DOI: https://doi.org/10.4049/jimmunol.188.Supp.43.21

Published: 01 May 2012

View article titled, A mixed Th1/Th2 response induces optimal resistance to Leishmania braziliensis (43.21)

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Journal Articles

Lymph Node Hypertrophy following Leishmania major Infection Is Dependent on TLR9

Lucas P. Carvalho, Patricia M. Petritus, Alyssa L. Trochtenberg, Colby Zaph, David A. Hill, David Artis, Phillip Scott

J Immunol (2012) 188 (3): 1394–1401.

DOI: https://doi.org/10.4049/jimmunol.1101018

Published: 01 February 2012

View article titled, Lymph Node Hypertrophy following Leishmania major Infection Is Dependent on TLR9

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The Early Generation of a Heterogeneous CD4⁺ T Cell Response to Leishmania major

Sara L. Colpitts, Phillip Scott

J Immunol (2010) 185 (4): 2416–2423.

DOI: https://doi.org/10.4049/jimmunol.1000483

Published: 15 August 2010

View article titled, The Early Generation of a Heterogeneous CD4+ T Cell Response to Leishmania major

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Journal Articles

IL-7 Receptor Expression Provides the Potential for Long-Term Survival of Both CD62L^high Central Memory T Cells and Th1 Effector Cells during Leishmania major Infection

Sara L. Colpitts, Nicole M. Dalton, Phillip Scott

J Immunol (2009) 182 (9): 5702–5711.

DOI: https://doi.org/10.4049/jimmunol.0803450

Published: 01 May 2009

View article titled, IL-7 Receptor Expression Provides the Potential for Long-Term Survival of Both CD62Lhigh Central Memory T Cells and Th1 Effector Cells during Leishmania major Infection

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Journal Articles

Interleukin-7 receptor (IL7R) expression provides the potential for long-term survival of both CD62L^high central memory T cells and Th1 effector cells during Leishmania major infection (96.6)

Sara L Colpitts, Nicole M Dalton, Phillip Scott

J Immunol (2009) 182 (1_Supplement): 96.6.

DOI: https://doi.org/10.4049/jimmunol.182.Supp.96.6

Published: 01 April 2009

View article titled, Interleukin-7 receptor (IL7R) expression provides the potential for long-term survival of both CD62Lhigh central memory T cells and Th1 effector cells during Leishmania major infection (96.6)

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Journal Articles

Functional Dichotomy of Dendritic Cells following Interaction with Leishmania braziliensis: Infected Cells Produce High Levels of TNF-α, whereas Bystander Dendritic Cells Are Activated to Promote T Cell Responses

Lucas P. Carvalho, Edward J. Pearce, Phillip Scott

J Immunol (2008) 181 (9): 6473–6480.

DOI: https://doi.org/10.4049/jimmunol.181.9.6473

Published: 01 November 2008

View article titled, Functional Dichotomy of Dendritic Cells following Interaction with Leishmania braziliensis: Infected Cells Produce High Levels of TNF-α, whereas Bystander Dendritic Cells Are Activated to Promote T Cell Responses

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Journal Articles

The Central Memory CD4⁺ T Cell Population Generated during Leishmania major Infection Requires IL-12 to Produce IFN-γ

Nazzy Pakpour, Colby Zaph, Phillip Scott

J Immunol (2008) 180 (12): 8299–8305.

DOI: https://doi.org/10.4049/jimmunol.180.12.8299

Published: 15 June 2008

View article titled, The Central Memory CD4+ T Cell Population Generated during Leishmania major Infection Requires IL-12 to Produce IFN-γ

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Journal Articles

Leishmania mexicana Infection Induces Impaired Lymph Node Expansion and Th1 Cell Differentiation Despite Normal T Cell Proliferation

Alice C. Hsu, Phillip Scott

J Immunol (2007) 179 (12): 8200–8207.

DOI: https://doi.org/10.4049/jimmunol.179.12.8200

Published: 15 December 2007

View article titled, Leishmania mexicana Infection Induces Impaired Lymph Node Expansion and Th1 Cell Differentiation Despite Normal T Cell Proliferation

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Journal Articles

STAT1 Expression in Dendritic Cells, but Not T Cells, Is Required for Immunity to Leishmania major

Leanne M. Johnson, Phillip Scott

J Immunol (2007) 178 (11): 7259–7266.

DOI: https://doi.org/10.4049/jimmunol.178.11.7259

Published: 01 June 2007

View article titled, STAT1 Expression in Dendritic Cells, but Not T Cells, Is Required for Immunity to Leishmania major

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Journal Articles

Antigen-Experienced T Cells Limit the Priming of Naive T Cells during Infection with Leishmania major

Peter M. Gray, Steven L. Reiner, Deborah F. Smith, Paul M. Kaye, Phillip Scott

J Immunol (2006) 177 (2): 925–933.

DOI: https://doi.org/10.4049/jimmunol.177.2.925

Published: 15 July 2006

View article titled, Antigen-Experienced T Cells Limit the Priming of Naive T Cells during Infection with Leishmania major

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Journal Articles

Persistence and Function of Central and Effector Memory CD4⁺ T Cells following Infection with a Gastrointestinal Helminth

Colby Zaph, Kathryn A. Rook, Michael Goldschmidt, Markus Mohrs, Phillip Scott, David Artis

J Immunol (2006) 177 (1): 511–518.

DOI: https://doi.org/10.4049/jimmunol.177.1.511

Published: 01 July 2006

View article titled, Persistence and Function of Central and Effector Memory CD4+ T Cells following Infection with a Gastrointestinal Helminth

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Journal Articles

In Vivo Recognition of Ovalbumin Expressed by Transgenic Leishmania Is Determined by Its Subcellular Localization

Sara Prickett, Peter M. Gray, Sara L. Colpitts, Phillip Scott, Paul M. Kaye, Deborah F. Smith

J Immunol (2006) 176 (8): 4826–4833.

DOI: https://doi.org/10.4049/jimmunol.176.8.4826

Published: 15 April 2006

View article titled, In Vivo Recognition of Ovalbumin Expressed by Transgenic Leishmania Is Determined by Its Subcellular Localization

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NARROW

Commensal microbiota induces IL-17A-producing innate lymphoid cells that augment skin inflammation in cutaneous leishmaniasis

A systemic inflammatory response primes blood monocytes in cutaneous leishmaniasis

MALNUTRITION IMPACTS PROTECTIVE IMMUNE RESPONSES AND MUCOSAL IMMUNITY DURING VISCERAL LEISHMANIASIS

CD8⁺ T Cells Lack Local Signals To Produce IFN-γ in the Skin during Leishmania Infection

Leishmania major Infection–Induced VEGF-A/VEGFR-2 Signaling Promotes Lymphangiogenesis That Controls Disease

Lymphocytic Choriomeningitis Virus Expands a Population of NKG2D⁺CD8⁺ T Cells That Exacerbates Disease in Mice Coinfected with Leishmania major

Bystander CD8 T cells produce granzyme B and promote exacerbated disease during Leishmania major infection (P3079)

Influence of viral specific memory cells on Leishmania major infection (164.12)

A mixed Th1/Th2 response induces optimal resistance to Leishmania braziliensis (43.21)

Lymph Node Hypertrophy following Leishmania major Infection Is Dependent on TLR9

The Early Generation of a Heterogeneous CD4⁺ T Cell Response to Leishmania major

IL-7 Receptor Expression Provides the Potential for Long-Term Survival of Both CD62L^high Central Memory T Cells and Th1 Effector Cells during Leishmania major Infection

Interleukin-7 receptor (IL7R) expression provides the potential for long-term survival of both CD62L^high central memory T cells and Th1 effector cells during Leishmania major infection (96.6)

Functional Dichotomy of Dendritic Cells following Interaction with Leishmania braziliensis: Infected Cells Produce High Levels of TNF-α, whereas Bystander Dendritic Cells Are Activated to Promote T Cell Responses

The Central Memory CD4⁺ T Cell Population Generated during Leishmania major Infection Requires IL-12 to Produce IFN-γ

Leishmania mexicana Infection Induces Impaired Lymph Node Expansion and Th1 Cell Differentiation Despite Normal T Cell Proliferation

STAT1 Expression in Dendritic Cells, but Not T Cells, Is Required for Immunity to Leishmania major

Antigen-Experienced T Cells Limit the Priming of Naive T Cells during Infection with Leishmania major

Persistence and Function of Central and Effector Memory CD4⁺ T Cells following Infection with a Gastrointestinal Helminth

In Vivo Recognition of Ovalbumin Expressed by Transgenic Leishmania Is Determined by Its Subcellular Localization

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