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A Bacterial and Ganglioside-based Nanoparticle Initiates Reprogramming of Macrophages and Promotes Antitumor Phenotypes

Rydell Alvarez-Arzola, Liliana Oliver, Michelle M. Messmer, Danielle Y.F. Twum, Kelvin P. Lee, Jason B. Muhitch, Circe Mesa, Scott I. Abrams

J Immunol (2024) 212 (3): 475–486.

https://doi.org/10.4049/jimmunol.2300256

Published: 20 December 2023

View articletitled, A Bacterial and Ganglioside-based Nanoparticle Initiates Reprogramming of Macrophages and Promotes Antitumor Phenotypes

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Includes: Supplementary data

Journal Articles

Multimodal Intralesional Therapy for Reshaping the Myeloid Compartment of Tumors Resistant to Anti–PD-L1 Therapy via IRF8 Expression

Ankit Patel, Takaaki Oba, Ryutaro Kajihara, Toshihiro Yokoi, Scott I. Abrams, Fumito Ito

J Immunol (2021) 207 (5): 1298–1309.

https://doi.org/10.4049/jimmunol.2100281

Published: 01 September 2021

View articletitled, Multimodal Intralesional Therapy for Reshaping the Myeloid Compartment of Tumors Resistant to Anti–PD-L1 Therapy via IRF8 Expression

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Includes: Supplementary data

Journal Articles

Seq-ing the source of radiation-induced anti-tumor immunogenicity in patient tumors

Jacky Louis Chow, Nicholas Hoffend, Madeline Gaudieri, Scott I Abrams, Thomas Schwaab, Anurag Singh, Jason Muhitch

J Immunol (2021) 206 (1_Supplement): 68.04.

https://doi.org/10.4049/jimmunol.206.Supp.68.04

Published: 01 May 2021

View articletitled, Seq-ing the source of radiation-induced anti-tumor immunogenicity in patient tumors

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Journal Articles

Radiation-Induced Immunity in Kidney Cancer Patients: Seq-ing Commonality Between Peripheral and Intratumoral T cell Repertoires

Jacky Louis Chow, Nicholas Hoffend, Scott I Abrams, Thomas Schwaab, Anurag Singh, Jason Bryan Muhitch

J Immunol (2020) 204 (1_Supplement): 241.27.

https://doi.org/10.4049/jimmunol.204.Supp.241.27

Published: 01 May 2020

View articletitled, Radiation-Induced Immunity in Kidney Cancer Patients: Seq-ing Commonality Between Peripheral and Intratumoral T cell Repertoires

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Journal Articles

Repression of osteopontin by IRF8 regulates a novel immunosuppressive checkpoint

John David Klement, Amy V Paschall, Priscilla S. Redd, Mohammed L. Ibrahim, Chunwan Lu, Dafeng Yang, Esteban Celis, Scott I Abrams, Keiko Ozato, Kebin Liu

J Immunol (2019) 202 (1_Supplement): 195.26.

https://doi.org/10.4049/jimmunol.202.Supp.195.26

Published: 01 May 2019

View articletitled, Repression of osteopontin by IRF8 regulates a novel immunosuppressive checkpoint

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Journal Articles

The Granulocyte Progenitor Stage Is a Key Target of IRF8-Mediated Regulation of Myeloid-Derived Suppressor Cell Production

Colleen S. Netherby, Michelle N. Messmer, Lauren Burkard-Mandel, Sean Colligan, Austin Miller, Eduardo Cortes Gomez, Jianmin Wang, Michael J. Nemeth, Scott I. Abrams

J Immunol (2017) 198 (10): 4129–4139.

https://doi.org/10.4049/jimmunol.1601722

Published: 15 May 2017

View articletitled, The Granulocyte Progenitor Stage Is a Key Target of IRF8-Mediated Regulation of Myeloid-Derived Suppressor Cell Production

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Includes: Supplementary data

Journal Articles

Entolimod, a clinical-stage TLR5 agonist, activates antitumor T cell immunity against liver and lung metastases through distinct mechanisms

Craig M Brackett, Bojidar Kojouharov, Kellee Greene, Nicholas Trageser, Sandra Gollnick, Lyudmila Burdelya, Scott I Abrams, Andrei Gudkov

J Immunol (2017) 198 (1_Supplement): 79.12.

https://doi.org/10.4049/jimmunol.198.Supp.79.12

Published: 01 May 2017

View articletitled, Entolimod, a clinical-stage TLR5 agonist, activates antitumor T cell immunity against liver and lung metastases through distinct mechanisms

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Journal Articles

Regulation of transcription factor IRF8 in myeloid progenitors is a critical checkpoint for formation of defective myeloid cells in cancer.

Michelle N Messmer, Colleen S Netherby, Scott I Abrams

J Immunol (2016) 196 (1_Supplement): 211.8.

https://doi.org/10.4049/jimmunol.196.Supp.211.8

Published: 01 May 2016

View articletitled, Regulation of transcription factor IRF8 in myeloid progenitors is a critical checkpoint for formation of defective myeloid cells in cancer.

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Journal Articles

The clinical-stage TLR5 agonist, Entolimod, mediates antimetastatic activity by stimulating a CXCR3-dependent NK-DC-CD8⁺ T cell axis

Craig M Brackett, Bojidar M. Kojouharov, Sandra O. Gollnick, Lyudmila G. Burdelya, Scott I Abrams, Andrei V. Gudkov

J Immunol (2016) 196 (1_Supplement): 75.5.

https://doi.org/10.4049/jimmunol.196.Supp.75.5

Published: 01 May 2016

View articletitled, The clinical-stage TLR5 agonist, Entolimod, mediates antimetastatic activity by stimulating a CXCR3-dependent NK-DC-CD8+ T cell axis

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Journal Articles

IRF8 represses GM-CSF expression in tumor cells to mediate myeloid-derived suppressor cell differentiation.

Amy V Paschall, Priscilla Redd, Ruihua Zhang, Huabao Xiong, Scott I Abrams, Kebin Liu

J Immunol (2016) 196 (1_Supplement): 211.10.

https://doi.org/10.4049/jimmunol.196.Supp.211.10

Published: 01 May 2016

View articletitled, IRF8 represses GM-CSF expression in tumor cells to mediate myeloid-derived suppressor cell differentiation.

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Journal Articles

IFN Regulatory Factor 8 Represses GM-CSF Expression in T Cells To Affect Myeloid Cell Lineage Differentiation

Amy V. Paschall, Ruihua Zhang, Chen-Feng Qi, Kankana Bardhan, Liang Peng, Geming Lu, Jianjun Yang, Miriam Merad, Tracy McGaha, Gang Zhou, Andrew Mellor, Scott I. Abrams, Herbert C. Morse, III, Keiko Ozato, Huabao Xiong, Kebin Liu

J Immunol (2015) 194 (5): 2369–2379.

https://doi.org/10.4049/jimmunol.1402412

Published: 01 March 2015

View articletitled, IFN Regulatory Factor 8 Represses GM-CSF Expression in T Cells To Affect Myeloid Cell Lineage Differentiation

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Includes: Supplementary data

Journal Articles

Modulating the Expression of IFN Regulatory Factor 8 Alters the Protumorigenic Behavior of CD11b⁺Gr-1⁺ Myeloid Cells

Trina J. Stewart, David J. Liewehr, Seth M. Steinberg, Kristy M. Greeneltch, Scott I. Abrams

J Immunol (2009) 183 (1): 117–128.

https://doi.org/10.4049/jimmunol.0804132

Published: 01 July 2009

View articletitled, Modulating the Expression of IFN Regulatory Factor 8 Alters the Protumorigenic Behavior of CD11b+Gr-1+ Myeloid Cells

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Journal Articles

Altered Immune Function during Long-Term Host-Tumor Interactions Can Be Modulated to Retard Autochthonous Neoplastic Growth

Trina J. Stewart, Scott I. Abrams

J Immunol (2007) 179 (5): 2851–2859.

https://doi.org/10.4049/jimmunol.179.5.2851

Published: 01 September 2007

View articletitled, Altered Immune Function during Long-Term Host-Tumor Interactions Can Be Modulated to Retard Autochthonous Neoplastic Growth

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Journal Articles

Cooperative downregulation of IFN[gamma]R and Fas is functionally linked to CTL-mediated immunoselection and tumor promotion (50.3)

Dafeng Yang, Kimberly K Smith, David Georgi, Scott I Abrams, Kebin Liu

J Immunol (2007) 178 (1_Supplement): S90.

https://doi.org/10.4049/jimmunol.178.Supp.50.3

Published: 01 April 2007

View articletitled, Cooperative downregulation of IFN[gamma]R and Fas is functionally linked to CTL-mediated immunoselection and tumor promotion (50.3)

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Journal Articles

LTbR Mediates Cell Contact-Dependent CTL-Directed Anti-Tumor Cytotoxicity (50.11)

Kebin Liu, Najam ud Din, Darren D Browing, Scott I Abrams, Dafeng Yang

J Immunol (2007) 178 (1_Supplement): S92.

https://doi.org/10.4049/jimmunol.178.Supp.50.11

Published: 01 April 2007

View articletitled, LTbR Mediates Cell Contact-Dependent CTL-Directed Anti-Tumor Cytotoxicity (50.11)

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Journal Articles

CTL Adoptive Immunotherapy Concurrently Mediates Tumor Regression and Tumor Escape

Kebin Liu, Sheila A. Caldwell, Kristy M. Greeneltch, Dafeng Yang, Scott I. Abrams

J Immunol (2006) 176 (6): 3374–3382.

https://doi.org/10.4049/jimmunol.176.6.3374

Published: 15 March 2006

View articletitled, CTL Adoptive Immunotherapy Concurrently Mediates Tumor Regression and Tumor Escape

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Journal Articles

Exposure of Human Primary Colon Carcinoma Cells to Anti-Fas Interactions Influences the Emergence of Pre-existing Fas-Resistant Metastatic Subpopulations

Kebin Liu, Elwood McDuffie, Scott I. Abrams

J Immunol (2003) 171 (8): 4164–4174.

https://doi.org/10.4049/jimmunol.171.8.4164

Published: 15 October 2003

View articletitled, Exposure of Human Primary Colon Carcinoma Cells to Anti-Fas Interactions Influences the Emergence of Pre-existing Fas-Resistant Metastatic Subpopulations

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Journal Articles

The Fas/Fas Ligand Pathway Is Important for Optimal Tumor Regression in a Mouse Model of CTL Adoptive Immunotherapy of Experimental CMS4 Lung Metastases

Sheila A. Caldwell, Mary H. Ryan, Elwood McDuffie, Scott I. Abrams

J Immunol (2003) 171 (5): 2402–2412.

https://doi.org/10.4049/jimmunol.171.5.2402

Published: 01 September 2003

View articletitled, The Fas/Fas Ligand Pathway Is Important for Optimal Tumor Regression in a Mouse Model of CTL Adoptive Immunotherapy of Experimental CMS4 Lung Metastases

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Journal Articles

Alterations in Fas Expression Are Characteristic of, But Not Solely Responsible for, Enhanced Metastatic Competence

Kebin Liu, Scott I. Abrams

J Immunol (2003) 170 (12): 5973–5980.

https://doi.org/10.4049/jimmunol.170.12.5973

Published: 15 June 2003

View articletitled, Alterations in Fas Expression Are Characteristic of, But Not Solely Responsible for, Enhanced Metastatic Competence

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Journal Articles

Irradiation of Tumor Cells Up-Regulates Fas and Enhances CTL Lytic Activity and CTL Adoptive Immunotherapy

Mala Chakraborty, Scott I. Abrams, Kevin Camphausen, Kebin Liu, Tamalee Scott, C. Norman Coleman, James W. Hodge

J Immunol (2003) 170 (12): 6338–6347.

https://doi.org/10.4049/jimmunol.170.12.6338

Published: 15 June 2003

View articletitled, Irradiation of Tumor Cells Up-Regulates Fas and Enhances CTL Lytic Activity and CTL Adoptive Immunotherapy

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A Bacterial and Ganglioside-based Nanoparticle Initiates Reprogramming of Macrophages and Promotes Antitumor Phenotypes

Multimodal Intralesional Therapy for Reshaping the Myeloid Compartment of Tumors Resistant to Anti–PD-L1 Therapy via IRF8 Expression

Seq-ing the source of radiation-induced anti-tumor immunogenicity in patient tumors

Radiation-Induced Immunity in Kidney Cancer Patients: Seq-ing Commonality Between Peripheral and Intratumoral T cell Repertoires

Repression of osteopontin by IRF8 regulates a novel immunosuppressive checkpoint

The Granulocyte Progenitor Stage Is a Key Target of IRF8-Mediated Regulation of Myeloid-Derived Suppressor Cell Production

Entolimod, a clinical-stage TLR5 agonist, activates antitumor T cell immunity against liver and lung metastases through distinct mechanisms

Regulation of transcription factor IRF8 in myeloid progenitors is a critical checkpoint for formation of defective myeloid cells in cancer.

The clinical-stage TLR5 agonist, Entolimod, mediates antimetastatic activity by stimulating a CXCR3-dependent NK-DC-CD8⁺ T cell axis

IRF8 represses GM-CSF expression in tumor cells to mediate myeloid-derived suppressor cell differentiation.

IFN Regulatory Factor 8 Represses GM-CSF Expression in T Cells To Affect Myeloid Cell Lineage Differentiation

Modulating the Expression of IFN Regulatory Factor 8 Alters the Protumorigenic Behavior of CD11b⁺Gr-1⁺ Myeloid Cells

Altered Immune Function during Long-Term Host-Tumor Interactions Can Be Modulated to Retard Autochthonous Neoplastic Growth

Cooperative downregulation of IFN[gamma]R and Fas is functionally linked to CTL-mediated immunoselection and tumor promotion (50.3)

LTbR Mediates Cell Contact-Dependent CTL-Directed Anti-Tumor Cytotoxicity (50.11)

CTL Adoptive Immunotherapy Concurrently Mediates Tumor Regression and Tumor Escape

Exposure of Human Primary Colon Carcinoma Cells to Anti-Fas Interactions Influences the Emergence of Pre-existing Fas-Resistant Metastatic Subpopulations

The Fas/Fas Ligand Pathway Is Important for Optimal Tumor Regression in a Mouse Model of CTL Adoptive Immunotherapy of Experimental CMS4 Lung Metastases

Alterations in Fas Expression Are Characteristic of, But Not Solely Responsible for, Enhanced Metastatic Competence

Irradiation of Tumor Cells Up-Regulates Fas and Enhances CTL Lytic Activity and CTL Adoptive Immunotherapy

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